Novel Mechanism Research on Neuropsychiatric Symptoms (NPS) in Alzheimer's Dementia (R01 Clinical Trial Optional)
This NIH R01 award funds research into the mechanisms behind neuropsychiatric symptoms in Alzheimer's disease and related dementias, with the goal of identifying novel therapeutic targets. It is ai…
- Deadline
- Sep 7, 2026
- Posted
- Nov 18, 2024
- Award amount
- Amount not specified
- Focus areas
- Health
In plain English
This NIH R01 award funds research into the mechanisms behind neuropsychiatric symptoms in Alzheimer's disease and related dementias, with the goal of identifying novel therapeutic targets. It is aimed at research institutions and investigators, with a broad list of eligible U.S. organization types. Award amounts are not specified here. Applications are submitted through eligible organizations rather than by individuals.
AI-generated summary to help you decide quickly — verify the official eligibility rules before applying.
Who can apply
Other Eligible Applicants include the following: Alaska Native and Native Hawaiian Serving Institutions; Asian American Native American Pacific Islander Serving Institutions (AANAPISISs); Eligible Agencies of the Federal Government; Faith-based or Community-based Organizations; Hispanic-serving Institutions; Historically Black Colleges and Universities (HBCUs); Indian/Native American Tribal Governments (Other than Federally Recognized); Non-domestic (non-U.S.) Entities (Foreign Organizations); Regional Organizations; Tribally Controlled Colleges and Universities (TCCUs) ; U.S. Territory or Possession.
About this grant
The goal of this Funding Opportunity Announcement (FOA) is to encourage applications for studies that will enhance knowledge of mechanisms associated with neuropsychiatric symptoms (NPS) in persons with Alzheimer's disease (AD) or Alzheimer's disease-related dementias (ADRD). The findings are expected to advance mechanistic understanding of both biobehavioral and neurobiological pathways leading to NPS. Findings may also provide insight into novel therapeutic targets that can be advanced into interventions to treat and prevent the development of NPS in AD and/or ADRD
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